Sierra Oncology Reports Preclinical Efficacy for Immunotherapy Combination with its Chk1 inhibitor SRA737

On November 29, 2018 Sierra Oncology, Inc. (Nasdaq: SRRA), a clinical stage drug development company focused on advancing targeted therapeutics for the treatment of patients with significant unmet needs in hematology and oncology, reported preclinical data for its novel oral Chk1 inhibitor, SRA737, in a poster presented at the American Association for Cancer Research (AACR) (Free AACR Whitepaper) Conference on Tumor Immunology and Immunotherapy in Miami Beach, Florida (Press release, Sierra Oncology, NOV 29, 2018, View Source [SID1234531745]).

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"In this study we found that the potent, selective oral Chk1 inhibitor, SRA737, activated the innate immune signaling STING pathway and demonstrated significant anti-tumor activity in an immunocompetent preclinical model of small cell lung cancer (SCLC)," said Dr. Lauren Byers, Associate Professor at the University of Texas MD Anderson Cancer Center, Houston, Texas. "Remarkably, the combination of SRA737 with an anti-PD-L1 immune checkpoint inhibitor induced tumor regression in this model, providing strong rationale for combining these agents to treat this immunotherapy refractory indication."

"SCLC remains a significant unmet need and one where immunotherapies have yielded limited efficacy. These tumors frequently harbor defects in cell cycle checkpoint and DNA damage repair (DDR) genes, with a coincident dependence on regulators of replication stress such as Checkpoint Kinase 1 (Chk1) as a compensatory mechanism," said Dr. Christian Hassig, Chief Scientific Officer, Sierra Oncology. "These encouraging preclinical results highlight an additional potential application for SRA737 that warrants further evaluation beyond our current clinical development programs for the drug as a monotherapy and in combination with replication stress-inducing low dose gemcitabine."

The poster will be presented on Thursday, November 29th from 5:00 to 7:00 pm (ET).

Title: The oral Chk1 inhibitor, SRA737, synergizes with immune checkpoint blockade in small cell lung cancer (SCLC).

Authors: Triparna Sen, Snezana Milutinovic, Robert J. Cardnell, Lixia Diao, Youhong Fan, Ryan J. Hansen, Bryan Strouse, Michael Hedrick, Christian Hassig, Jing Wang, Lauren A. Byers.

Location: Poster Session: B; Board Number: B15; Session Location: Americana 3 and 4

STING: Stimulator of Interferon Genes

About SRA737
SRA737 is a potent, highly selective, orally bioavailable small molecule inhibitor of Checkpoint kinase 1 (Chk1), a key regulator of cell cycle progression and the DNA Damage Response (DDR). In cancer cells, intrinsic replication stress is induced by factors such as oncogenes (e.g., CCNE1 or MYC), genetic mutations in DNA repair machinery (e.g., BRCA1 or FANCA), genetic mutations leading to a dysregulated cell cycle (e.g., TP53 or RAD50) or other genomic alterations. This replication stress results in persistent DNA damage and genomic instability leading to an increased dependency on Chk1 for survival. Targeted inhibition of Chk1 by SRA737 may therefore be synthetically lethal to cancer cells with elevated intrinsic replication stress and have utility as a monotherapy in a range of tumor indications. The combination of SRA737 with other modalities, such as other agents that target the DDR network and certain chemotherapeutics, may also provide synergistic anti-tumor activity via a variety of potential biological mechanisms. Importantly, the oral bioavailability of SRA737 may afford greater dosing flexibility for both monotherapy and combination therapy settings than is possible with intravenously administered agents.

SRA737 is currently being investigated in two Phase 1/2 clinical trials primarily focused on patients with ovarian cancer: SRA737-01, a monotherapy study, and SRA737-02, a drug combination study evaluating SRA737 potentiated by low dose gemcitabine. Sierra has also prepared for a potential clinical study of SRA737 in combination with a PARP inhibitor.

Constellation Pharmaceuticals to Present at BMO Healthcare Conference

On November 29, 2018 Constellation Pharmaceuticals, Inc. (Nasdaq: CNST), a clinical-stage biopharmaceutical company using its expertise in epigenetics to discover and develop novel therapeutics, reported that Jigar Raythatha, CEO, will present at the BMO 2018 Prescriptions for Success Healthcare Conference at the Mandarin Oriental Hotel in New York at 10:40 AM on Wednesday, December 12 (Press release, Constellation Pharmaceuticals, NOV 29, 2018, View Source [SID1234531744]). A live audio webcast of the presentation and an archive for replay will be available on the Investor Relations section of Constellation’s website at View Source The audio webcast replay will be available for 90 days following the live presentation.

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Atara Biotherapeutics Highlights Next-Generation and Off-the-Shelf, Allogeneic CAR T Technologies and Pipeline

On November 29, 2018 Atara Biotherapeutics, Inc. (Nasdaq: ATRA), a leading off-the-shelf, allogeneic T-cell immunotherapy company developing novel treatments for patients with cancer, autoimmune and viral diseases, reported details of the Company’s next-generation chimeric antigen receptor T cell (CAR T) technologies and pipeline in conjunction with hosting a CAR T Breakfast Teach-In event in New York, NY (Press release, Atara Biotherapeutics, NOV 29, 2018, View Source [SID1234531739]).

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"Atara is building a leading next-generation and off-the-shelf, allogeneic CAR T pipeline," said Isaac Ciechanover M.D., Chief Executive Officer and President of Atara Biotherapeutics. "Although the current generation autologous CAR T immunotherapies have transformed outcomes for patients with B cell malignancies, we believe many opportunities exist to improve outcomes, expand to earlier lines of therapy and address areas of high unmet need, including acute myelogenous leukemia (AML) and solid tumors. We are collaborating with academic leaders to advance programs using technologies at the forefront of CAR T immunotherapy innovation."

The event featured next generation CAR T experts:

Michel Sadelain, M.D., Ph.D., Director, Center for Cell Engineering, and Head, Gene Expression and Gene Transfer Laboratory, Memorial Sloan Kettering Cancer Center (MSK)
Marco Davila, M.D., Ph.D., Associate Attending Physician, Department of Blood & Marrow Transplantation and Cellular Immunotherapy Medical Director, Cell Therapy Facility, Moffitt Cancer Center
Dr. Sadelain described CAR T technologies including novel co-stimulatory domains modified to generate more physiologic signaling, reduce exhaustion, improve persistence and unlock the solid tumor microenvironment. Atara and MSK are collaborating to develop next-generation CAR T immunotherapies using several of these innovative CAR T technologies.

Dr. Davila’s presentation focused on developing promising CAR T immunotherapies for patients with AML and B cell malignancies in collaboration with Atara using modified co-stimulatory subdomains designed to improve persistence and reduce T cell exhaustion.

Dietmar Berger, M.D., Ph.D., Global Head of Research & Development of Atara Biotherapeutics introduced the Company’s strategy to leverage these innovative licensed technologies with Atara’s EBV-specific T cell expertise to advance next-generation, off-the-shelf CAR T programs.

Dr. Berger presented results for an EBV.CD19.28z CAR T that demonstrated high CAR transduction, increased frequency of central memory T cell phenotype, specific and selective CD19 activity, low levels of off-target alloreactivity and strong antigen-specific proliferation and persistence. These findings establish feasibility for engineering EBV-specific T cells by leveraging next-generation CAR technologies, and support further development as an off-the-shelf, allogeneic, CAR T platform to generate IND clinical candidates. He concluded by describing Atara’s CAR T preclinical pipeline, including four oncology programs progressing toward IND.

A webcast replay of the Atara CAR T Breakfast Teach-In can be accessed on the Investors and Media section of Atarabio.com for 30 days following today’s live event.

NOX Announces Positive Data from CEP-1 Study of Veyonda

On November 29, 2018 Noxopharm (ASX: NOX) reported the final results from its Phase 1b CEP-1 trial evaluating its lead immuno-oncology/radio-enhancer drug candidate, Veyonda, in combination with low-dose carboplatin in late-stage metastatic solid cancers (Press release, Noxopharm, NOV 29, 2018, View Source [SID1234531738]).

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The CEP-1 trial was a first-in-human study for Veyonda and was designed to confirm the safety of Veyonda both as a monotherapy and in combination with chemotherapy, as well as patient and doctor acceptance of the treatment regimen. The study, which ran between January 2017 and July 2018, was managed by a UK-based clinical research organisation and conducted in the European country of Georgia at sites subject to FDA audit. Top line data was presented earlier at the Clinical Oncology Society of Australia (COSA) Annual Meeting on 13 November 2018. With the Final Report now received, the Company is able to comment on the outcome, in particular, the effect of treatment on cancer progression.

The topline outcomes of the study are:

Veyonda was well tolerated as a monotherapy, with just 1 case of anemia attributed to it
Veyonda at 400mg and 800 mg dosages did not exacerbate carboplatin toxicity
A combination of Veyonda and low-dose carboplatin provided suspension of tumour growth, or better, for at least 6 months in solid tumours (breast, ovarian, lung, prostate) in nearly 50% of patients considered unlikely to respond to further chemotherapy
The Company believes that the response rate and ongoing survival of patients post-study, suggests that a meaningful increase in survival is achievable and therefore worthy of consideration of an eventual marketing approval process
The Company also believes that this points to a combination of Veyonda and low-dose carboplatin as offering a potential treatment option for those cancer patients considered too unwell to undergo chemotherapy and/or unlikely to respond to chemotherapy because of their cancer becoming unresponsive to chemotherapy.
Study details. CEP-1 recruited 19 subjects with late-stage metastatic solid cancers (breast, ovarian, lung, prostate) who had stopped responding to chemotherapy, including carboplatin, and for whom no remaining standard treatment options were available. All patients entered the study with progressive disease and were assessed as having limited survival prospects. Subjects were administered Veyonda as a monotherapy for the first month and then in combination with two dosages of carboplatin (3 cycles of carboplatin followed by 3 cycles of carboplatin at 50% and 75% respectively of a standard dose) over six months, producing a 7-month treatment course in full.

Safety: With the exception of one (1) patient who displayed hypersensitivity to carboplatin at the initial injection, in no case was toxicity severe enough to require combination therapy needing to be withheld or have the dosage reduced.

Overall, 83% of patients experienced 1 or more adverse events, the majority during combination therapy. The most common events were anaemia (low red blood cells), neutropenia (low white blood cells) and hypocalcemia (low blood calcium levels). 95% of events occurred during combination therapy, of which 80% were attributed to carboplatin. One (1) severe case of anaemia was reported during monotherapy and possibly attributed to Veyonda.

Cancer status: A high proportion of patients showed tumour responses ranging from stable disease (a halt in disease progression) to partial response (decreased tumour load), despite a patient population that was heavily pre-treated with chemotherapy and assessed as ineligible for further treatment. Individual efficacy results for patients assessable after 3 cycles of combination therapy (left figure) and after 6 cycles (right figure) are detailed in the figure below. Bars reflect the change in total tumor(s) size for each patient and annotations show categorical response according to RECIST 1.1 criteria (PR = partial response; SD = stable disease; PD = progressive disease) for the same patients. Fourteen patients completed 3 cycles and were able to be assessed; 8 completed 6 cycles and were able to be assessed.

At 3-months, 10 of 14 patients had shown no tumour progression; 8 of these were evaluable after a further 3 cycles (6 cycles total) and of these, 6 patients had shown no disease progression at the end of the study, including one partial responder with an almost 100% reduction in tumour size.

Two photos accompanying this announcement are available at
View Source
View Source

Rationale: The active ingredient in Veyonda, idronoxil, restores sensitivity to carboplatin in cancer cells that have stopped responding to carboplatin after multiple courses of treatment. The rationale of the CEP program is that Veyonda will restore sensitivity to chemotherapy or will act synergistically with chemotherapy, such that the dosage of chemotherapy can be lowered to a tolerable level, if required. The ultimate objective is to utilize Veyonda as a chemo-sensitizer to provide meaningful anti-cancer effects while avoiding serious side-effects that are often treatment-limiting, particularly in in patients considered too unwell to undergo cancer therapy. The CEP regimen also offers the prospect of improving tolerability of chemotherapy regimens in children, where successful treatment with chemotherapy can come at the cost of toxicities with long-term sequelae.

Comments: "The CEP-1 data are highly encouraging, suggesting that in addition to serving as a potential enhancer of radiotherapy, Veyonda appears to have broader utility as an enhancer of chemotherapy as well," said Greg van Wyk M.D., Noxopharm Chief Medical Officer. "Chemotherapy-induced toxicity remains a significant challenge for patients and oncologists that can lead to long-lasting and debilitating side effects, such as peripheral nerve damage and hearing loss. We are hopeful that combining Veyonda with a lower than normal dosage of chemotherapy will provide a more tolerable treatment option for patients who have chemotherapy-resistant disease or who are only able to tolerate lower doses of chemotherapy."

Graham Kelly Ph.D., Noxopharm Chief Executive Officer, said, "We believe that this result goes a considerable way to confirming the good tolerability and efficacy of Veyonda. A high incidence of an anti-cancer effect in such a highly treatment-resistant patient population is very pleasing. The context here is that these patients had progressive disease, had exhausted standard treatment options, and were facing a limited lifespan. To be able to stop tumour growth or to even shrink the tumours in many of these patients over the 7 months of this study, and to do so without significant side-effects, substantiates the faith we have in this drug becoming an important addition to standard anti-cancer therapy."

"Noxopharm is committed to bringing Veyonda to market as a radio-enhancer with a strategy that we believe will see it on-market by 2022, but the CEP-1 outcome adds another dimension to our overall clinical and marketing strategies," Kelly added.

The Company now will consult with its advisors on a Phase 2 CEP study in Australian cancer patients, although its primary focus remains on its DARRT and LuPIN programs as being the likely quickest routes to market.

A family of three patents surrounding Veyonda, including one pertaining to use with chemotherapy, has entered the global national phase following a review by the International Examiner.

About Veyonda

Veyonda (previously known as NOX66) is an innovative dosage formulation of the experimental anti-cancer drug, idronoxil, developed specifically to preserve the anti-cancer activity of idronoxil in the body and to enhance its drug-like behaviour. Idronoxil is a kinase inhibitor that works by inhibiting a range of enzymes, pre-eminent among which is sphingosine kinase, a key regulator of cell pro-survival mechanisms, and which is over-expressed in many cancer cells. Idronoxil also is an immuno-oncology drug, activating the body’s innate immune system e.g. natural killer (NK) cells.

About CEP

The Company’s CEP Program (Chemotherapy Enhancement Program) is testing the ability of Veyonda to restore sensitivity of cancer cells to carboplatin in patients whose late-stage cancers have stopped responding to chemotherapy, and to do that to the extent that the dosage of carboplatin can be lowered to a level unlikely to cause serious adverse side-effects. The clinical outcome being sought is the ability to offer a well-tolerated chemotherapy regimen to patients considered unsuitable for standard dosage due to age or illness.

RedHill Biopharma to Present at BMO 2018 Prescriptions for Success Healthcare Conference

On November 29, 2018 RedHill Biopharma Ltd. (Nasdaq: RDHL) (Tel-Aviv Stock Exchange: RDHL) ("RedHill" or the "Company"), a specialty biopharmaceutical company primarily focused on proprietary drugs for gastrointestinal diseases, reported that Mr. Dror Ben-Asher, chief executive officer of RedHill, will present a corporate overview at the BMO 2018 Prescriptions for Success Healthcare Conference on Wednesday, December 12, 2018, at 3:00 p.m. EST at the Mandarin Oriental Hotel, New York (Press release, RedHill Biopharma, NOV 29, 2018, View Source [SID1234531735]).

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The presentation will be broadcast live and available via replay for 30 days on the Company’s website, View Source Please access the website at least 15 minutes ahead of the conference call to register.