Biomunex Announces the Upcoming Start of Clinical Development of IPN60330, the First MAIT Engager, a New Therapeutic Class in Oncology, Following the Completion of the FDA Review Period

On September 14, 2026 Biomunex Pharmaceuticals, a biopharmaceutical company specialized in the discovery and development of innovative therapeutic approaches, reported that the U.S. Food and Drug Administration (FDA) has completed its Investigational New Drug (IND) review period for Ipsen’s IPN60330 (formerly BMX-502), the first MAIT cell engager (also referred to as a "MAIT engager") based on Biomunex’s proprietary BiXAb technology platforms, and for which Biomunex and Ipsen announced an exclusive global licensing agreement for development, manufacture and commercialization rights in 2024.

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This key step is the result of an intensive preclinical development effort, encompassing full drug bioproduction for the Phase 1 trial, regulatory studies and animal toxicology studies, conducted by the Biomunex team in partnership with Ipsen. It marks the upcoming initiation of Ipsen’s Phase I clinical trial in the USA, a significant step forward in the development of this novel therapeutic class in immuno-oncology.

IPN60330 is a bispecific antibody designed to selectively engage and activate MAIT cells (Mucosal-Associated Invariant T cells), a subset of T cells present throughout the body, which are particularly enriched in mucosal and barrier tissues, and targets the GPC3 tumor antigen to kill cancer cells. GPC3 is a clinically validated target, highly expressed across several cancer types.

The completion of the FDA review period is a further demonstration of the quality of Biomunex’s proprietary BiXAb technologies. This first MAIT engager should overcome some of the limitations of classical pan-T cell engagers, which activate all T cells, including regulatory T cells, and can induce cytokine release syndrome, a potentially serious dose-limiting toxicity that represents a true problem for physicians and their cancer patients. MAIT engagers have the potential to provide a broader therapeutic window compared to classical pan-T cell engagers for treating specific tumor types. Moreover, an improved therapeutic window with MAIT engagers should permit a stronger response from the adaptive immune system leading to improved and durable responses.

This new milestone has triggered a milestone payment linked to the completion of the FDA IND review period ahead of the upcoming entry into clinical development, under the exclusive global licensing agreement entered into between Ipsen and Biomunex in November 2024. In the context of this agreement, Biomunex is eligible to receive up to $610 million in total, including an upfront already received, contingent upon successful development, regulatory and commercial milestones, in addition to tiered global royalties on sales.

"The upcoming start of the clinical development program in the United States for IPN60330 is a recognition of the quality of our collaboration with Ipsen. Moreover, that’s a major milestone in our development, demonstrating the strength of our BiXAb technology platforms, as well as the expertise of our teams in generating breakthrough innovation," said Dr Simon Plyte, CSO of Biomunex. "It finally underscores our ability to develop innovative programs in partnership with a world-renowned pharmaceutical partner, with the potential to open new avenues in cancer immunotherapy," added Dr Pierre-Emmanuel Gerard, founder, President and CEO of Biomunex.

(Press release, BIOMUNEX Pharmaceuticals, SEP 14, 2026, View Source [SID1234670843])

Shennon Biotechnologies Raises $12M in Financing, Appoints Cyril Konto, M.D., as Chief Executive Officer and Unveils Precision Immunotherapy Pipeline

On September 14, 2026 Shennon Biotechnologies ("ShennonBio"), a biotechnology company developing precision immunotherapies for cancer, reported that it has raised $12M in financing from Future Ventures, NextGen Venture Partners, Samos Investments, Atypical Ventures and Saras Capital, with continued support from existing investors DCVC, Foundation Capital, and AV8 Ventures. The company has raised $25M to date.

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In addition to the funding, the Company has appointed biotechnology industry veteran Cyril Konto, M.D. as Chief Executive Officer. Founder and current CEO Li Sun, Ph.D., will transition to President and Chief Technology Officer, continuing to lead the Company’s scientific strategy, technology development and pipeline innovation.

The new financing will support continued development of ShennonBio’s integrated technology platform and advancement of its emerging oncology pipeline that currently includes three T-cell engager (TCE) programs in small cell lung cancer, ovarian cancer and hepatocellular carcinoma.

Since its founding in 2021, ShennonBio has built a proprietary technology engine spanning target discovery, ultra-high-throughput functional screening, computational binder prediction and optimization, and AI-enabled safety assessment. The Company has also established approximately 6,000 square feet of laboratory space in San Francisco near UCSF Mission Bay and assembled proprietary datasets with fresh tumor samples from strong partnerships with hospitals and academic institutions.

"ShennonBio was founded to address fundamental challenges that have limited the development of precision immunotherapies, from identifying differentiated targets and highly functional therapeutic binders to predicting potential safety risks before candidates reach the clinic," said Li Sun, Ph.D., Founder, President and Chief Technology Officer, ShennonBio. "We have built an integrated technology platform, generated proprietary human datasets and begun translating those capabilities into a pipeline of precision immunotherapies. With $25 million raised to date, and Cyril joining as CEO, we are entering an important new stage for ShennonBio as we advance our first TCE programs toward the clinic."

Building an Integrated Platform for Precision Immunotherapy

ShennonBio is developing both antibody-based T-cell engagers (Ab-TCEs), which can address targets expressed on the surface of tumor cells, and TCR-based T-cell engagers (TCR-TCEs), which can potentially access intracellular cancer targets presented through HLA. This dual approach is designed to substantially expand the universe of cancer biology that can potentially be addressed through T-cell engagers.

The Company’s integrated platform combines four proprietary capabilities:

PINTRA, which uses proprietary human tumor datasets and advanced computational approaches to identify differentiated surface and intracellular therapeutic targets;
TCELERATOR, an ultra-high-throughput functional screening platform capable of evaluating more than 10 million single-cell interactions concurrently and millions of therapeutic candidates;
AFFINIS, a computational platform for predicting, designing and optimizing antibody binders and TCR; and
SERIS, an AI-enabled safety platform designed to identify potential off-target cross-reactivity earlier in therapeutic development.
Together, these technologies create an integrated discovery and development engine spanning target identification, functional screening, therapeutic optimization and safety assessment.

A central component of ShennonBio’s approach is its growing proprietary human dataset. The Company has assembled data encompassing >10,000 tumor samples, >11 million single cells and a substantial collection of TCR sequences. ShennonBio continues to expand these datasets through relationships involving more than 15 institutions and across multiple cancer types and other diseases.

Advancing a Diversified Oncology Pipeline

ShennonBio has translated its platform into three initial oncology programs spanning both antibody- and TCR-based T-cell engagers.

SBT-121 is a dual-targeting trispecific Ab-TCE being developed for small cell lung cancer.

SBT-201 is an Ab-TCE being developed for ovarian cancer.

SBT-425 is a TCR-TCE being developed for hepatocellular carcinoma.

Together, the programs reflect ShennonBio’s strategy of combining established T-cell engager biology with its proprietary discovery and screening capabilities to pursue differentiated therapeutic opportunities across solid tumors.

As ShennonBio advances from platform development toward development-stage execution, the Company has appointed Cyril Konto, M.D., as Chief Executive Officer. Dr. Sun, ShennonBio’s founder and current CEO, will transition to President and Chief Technology Officer, and will remain deeply committed to the Company, leading scientific strategy, technology development and pipeline innovation.

Dr. Konto brings more than 20 years of experience in oncology drug development, company building and strategic transactions. Most recently, he served as President and Chief Executive Officer of Ichnos Glenmark Innovation (IGI), where he led a strategic and operational transformation of the organization, focused its portfolio on oncology and built its multispecifics franchise.

During his tenure, IGI completed three external partnerships, including an agreement with AbbVie for a Phase 1 trispecific T-cell engager that included $700 million upfront and up to $1.9 billion in total potential value plus royalties. He previously held senior oncology development leadership roles at Allogene Therapeutics, Pfizer and Bristol Myers Squibb.

"ShennonBio has spent the last several years building a differentiated foundation spanning proprietary human data, target discovery, functional screening, therapeutic design and safety prediction," said Dr. Konto. "What attracted me to the Company is the opportunity to translate those capabilities into a new generation of precision immunotherapies. I look forward to working closely with Li and the team to advance our pipeline toward the clinic, establish strategic partnerships and build ShennonBio for its next stage of growth."

Over the next two years, ShennonBio plans to advance a development candidate toward IND filing and Phase 1 clinical development, pursue licensing opportunities for selected therapeutic programs and explore partnerships around its platform capabilities, including binder prediction and generation, clinical toxicity prediction and its proprietary datasets.

(Press release, Shennon Biotechnologies, SEP 14, 2026, View Source [SID1234670842])

Vyriad Begins Phase 1 Clinical Trial for Lead In Vivo CAR T Cell Therapy VV169 Following FDA Acceptance of IND Application

On September 14, 2026 Vyriad, Inc., a clinical-stage biotechnology company developing targeted genetic therapies for cancer and other serious diseases, reported that the U.S. Food and Drug Administration (FDA) has cleared its Investigational New Drug (IND) application to initiate a clinical trial of VV169, an off-the-shelf, in vivo CAR T cell therapy targeting BCMA in patients with relapsed/refractory multiple myeloma. The Phase 1 trial is now open for enrollment at Mayo Clinic.

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"VV169 is designed to create CAR-T cells directly inside the body, offering a new way of delivering this powerful therapeutic approach to patients with multiple myeloma. The Phase 1 clinical trial for VV169 being open for enrollment is a significant milestone for Vyriad and we are proud to reach it alongside Dr. Yi Lin and her team at Mayo Clinic," said Luke Russell, Ph.D., MBA, President of Vyriad. "This reflects years of sustained effort from investigators, study staff, and the R&D and manufacturing teams here in Rochester. We are grateful to everyone who made it happen, and to the patients who choose to take part."

VV169 was developed using Vyriad’s proprietary in vivo lentiviral platform centered on blinded and retargeted VSV-G pseudotyping. To enable direct in vivo delivery, VV169 utilizes the targeted lentiviral vector to deliver a potent anti-BCMA CAR payload. Upon intravenous administration, the vehicle specifically targets and transduces T cells, enabling rapid generation of functional CAR T cells. This targeted approach results in highly specific payload expression and minimizes the risk of infusional toxicities.

"For patients with aggressive multiple myeloma, waiting weeks for a therapy to be manufactured can be a significant barrier," said Chief Scientific Officer Stephen Russell, M.D., Ph.D. "VV169 is designed to change that by creating CAR T cells directly inside the body, thereby making this off-the-shelf approach to CAR T more accessible for patients. We look forward to evaluating the potential of this program in the clinic."

The Phase 1 trial will evaluate the safety and initial efficacy signals of VV169 in up to 40 patients with relapsed/refractory multiple myeloma. The trial, conducted at Mayo Clinic by principal investigator Yi Lin, M.D., Ph.D., head of the center’s in vivo CAR T program, will have key primary objectives to assess overall safety, identify dose-limiting toxicities, and determine the recommended Phase 2 dose. The secondary objective will focus on assessing preliminary efficacy signals of VV169 and evaluating in vivo CAR T cell expansion and persistence.

For more information, including trial details and eligibility criteria, please visit the study page on clinicaltrials.gov (NCT07802717).

(Press release, Vyriad, SEP 14, 2026, View Source [SID1234670841])

MaaT Pharma Receives Negative CHMP Trend Vote for MaaT013 (Xervyteg®) Following Re-Examination for its Conditional Marketing Authorization Application in Europe

On September 14, 2026 MaaT Pharma (EURONEXT: MAAT – the "Company"), a clinical-stage biotechnology company and a leader in the development of Microbiome Ecosystem Therapies (MET) dedicated to enhancing survival for patients with cancer through immune modulation, reported that it has been informed by the CHMP of the European Medicines Agency (EMA) of a "negative trend" opinion following the Oral Explanation, as part of the re-examination procedure of its conditional Marketing Authorization Application (MAA) for MaaT013 (Xervyteg) for the treatment of aGvHD, following yesterday’s CHMP oral explanation.

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"We are deeply disappointed by this outcome. Patients suffering from gastrointestinal-aGvHD continue to face a life-threatening condition with significant unmet medical need and limited treatment options," said Hervé Affagard, Chief Executive Officer and co-founder of MaaT Pharma. "Together with the hematology community, we remain committed to these patients and will evaluate all available options to make this potentially important treatment accessible to patients worldwide."

Based on the feedback received post Oral Explanation, the CHMP maintained its view that the available clinical data package, primarily in the absence of a randomized control trial, does not allow sufficient characterization of the benefit/risk of MaaT013 (Xervyteg).

The Company will provide additional details following the formal CHMP opinion, which is expected to be communicated on September 18, 2026. The Company will assess the implications of the decision and evaluate all available options and will provide an update to the market as appropriate.

(Press release, MaaT Pharma, SEP 14, 2026, View Source [SID1234670840])

FDA APPROVES REDUCED MONITORING TIME FOR FIRST TWO DOSES OF IMDELLTRA

On September 14, 2026 Amgen (NASDAQ:AMGN) reported that the U.S. Food and Drug Administration (FDA) approved an update to the IMDELLTRA (tarlatamab-dlle) Prescribing Information that substantially reduces the recommended monitoring time for the first two doses of treatment in an appropriate healthcare setting.

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This approval means that patients receiving IMDELLTRA should now be monitored for 6 to 8 hours from the start of their first two infusions, compared with the previously recommended 22 to 24 hours. Patients will also receive a follow-up assessment, including vital signs, the day after each of these first two doses.

For people living with extensive-stage small cell lung cancer (ES-SCLC), the change could mean substantially less time spent in a healthcare setting during the initial treatment period. The updated requirement may also reduce treatment complexity for community oncology practices, where an estimated 80% to 85% of U.S. cancer patients receive care.1

"People being treated for small cell lung cancer are already navigating an aggressive and difficult-to-treat disease, and the time required to receive and monitor treatment can add to that burden for both patients and care centers," said Jay Bradner, M.D., executive vice president, Research and Development, Artificial Intelligence and Data at Amgen. "This approval is an important step in simplifying care for people living with and treating ES-SCLC. Reducing monitoring to 6-8 hours for the initial doses may help address practical barriers associated with administering IMDELLTRA and enable more patients to receive care closer to home. We continue to work closely with the healthcare community to ensure appropriate educational support for navigating IMDELLTRA treatment is available to providers, patients and care partners."

"In community oncology, we care for patients where they live, and for people living with small cell lung cancer, that matters," said David M. Waterhouse, MD, MPH, FASCO, medical oncologist/hematologist at Oncology Hematology Care in Cincinnati, OH. "These patients are often very sick, and traveling long distances or spending extended time in a healthcare setting can be difficult for them and their families. Reducing the required monitoring period may make IMDELLTRA more feasible to administer in community practices, helping more patients receive treatment in their local care network and spend less time away from their support systems."

ES-SCLC is an aggressive form of lung cancer in which most patients experience disease progression following first-line treatment.2 The scope of the label update was limited to the first two doses, which now recommends only 6 to 8 hours of monitoring from the start of infusion in an appropriate healthcare setting. Beyond the update to the first two doses and the addition of a patient assessment following those initial doses, the monitoring and supportive care recommendations remain unchanged: 6-8 hours of monitoring following the third dose (Cycle 1 Day 15) and throughout Cycle 2, decreasing to 3-4 hours for Cycles 3-4 and 2 hours for Cycle 5 and subsequent doses. It is recommended that patients remain within 1 hour of an appropriate healthcare setting for a total of 48 hours from the start of the infusion with IMDELLTRA following Cycle 1 Day 1 and Cycle 1 Day 8 doses, accompanied by a caregiver. Patients will also receive a follow-up assessment, including vital signs, the day after each of these first two doses on Cycle 1, Days 2 and 9.

The updated monitoring requirements represent an important evolution in the administration of IMDELLTRA and reinforce Amgen’s commitment to advancing treatments while addressing the real-world needs of people living with cancer.

Amgen recently announced landmark positive topline overall survival data from the Phase 3 DeLLphi-305 study in first-line ES-SCLC. Reduced post-infusion monitoring timing is being evaluated in several ongoing studies across indications and lines of therapy which will continue to inform the potential to further reduce monitoring approaches in the future.

About Small Cell Lung Cancer (SCLC)
SCLC is one of the most aggressive and devastating forms of solid tumor cancer. Each year, SCLC accounts for approximately 13-15% of more than 2.6 million cases of lung cancer diagnosed worldwide.2-4 Despite initial high response rates to first-line platinum-based chemotherapy, most patients quickly relapse within months and require subsequent treatment options.2

About IMDELLTRA (tarlatamab-dlle)
IMDELLTRA is a first-in-class targeted immunotherapy engineered by Amgen researchers to bind to both DLL3 on tumor cells and CD3 on T cells, thereby activating T cells to kill DLL3-expressing SCLC cells. This results in the formation of a cytolytic synapse with lysis of the cancer cell.5,6 DLL3 is a protein that is expressed on the surface of SCLC cells in ~85-96% of patients with SCLC, but is minimally expressed on healthy cells, making it an exciting target.7,8

U.S. INDICATION
IMDELLTRA (tarlatamab-dlle) is indicated for the treatment of adult patients with extensive stage small cell lung cancer (ES-SCLC) with disease progression on or after platinum-based chemotherapy.

IMPORTANT SAFETY INFORMATION

WARNING: CYTOKINE RELEASE SYNDROME and NEUROLOGIC TOXICITY including IMMUNE EFFECTOR CELL-ASSOCIATED NEUROTOXICITY SYNDROME

Cytokine release syndrome (CRS), including life-threatening or fatal reactions, can occur in patients receiving IMDELLTRA. Initiate IMDELLTRA using the step-up dosing schedule to reduce the incidence and severity of CRS. Withhold IMDELLTRA until CRS resolves or permanently discontinue based on severity.
Neurologic toxicity and immune effector cell-associated neurotoxicity syndrome (ICANS), including life-threatening or fatal reactions, can occur in patients receiving IMDELLTRA. Monitor patients for signs and symptoms of neurologic toxicity, including ICANS, during treatment and treat promptly. Withhold IMDELLTRA until ICANS resolves or permanently discontinue based on severity.
WARNINGS AND PRECAUTIONS

Cytokine Release Syndrome (CRS): IMDELLTRA can cause CRS including life-threatening or fatal reactions. In the pooled safety population, CRS occurred in 57% (268/473) of patients who received IMDELLTRA, including 39% Grade 1, 15% Grade 2, 1.7% Grade 3 and 0.2% Grade 4. Recurrent CRS occurred in 24% of IMDELLTRA-treated patients including 20% Grade 1 and 3.4% Grade 2; one patient experienced recurrent Grade 3.

Among the 268 patients who experienced CRS, 73% had CRS after the first dose, 60% had CRS after the second dose, and 15% had CRS following the third or later dose. Following the Cycle 1 Day 1, Day 8, Day 15 infusions, 24%, 8%, and 1% of patients experienced Grade ≥ 2 CRS, respectively. From Cycle 2 onwards, 1.5% of patients experienced Grade ≥ 2 CRS. Of the patients who experienced CRS, 31% received steroids and 10% required tocilizumab. The median time to onset of all grade CRS from most recent dose of IMDELLTRA was 16 hours (range: start of infusion to 15 days). The median time to onset of Grade ≥ 2 CRS from most recent dose of IMDELLTRA was 15 hours (range: start of infusion to 15 days).

Clinical signs and symptoms of CRS included pyrexia, hypotension, fatigue, tachycardia, headache, hypoxia, nausea, and vomiting. Potentially life-threatening complications of CRS may include cardiac dysfunction, acute respiratory distress syndrome, neurologic toxicity, renal and/or hepatic failure, and disseminated intravascular coagulation (DIC).

Administer IMDELLTRA following the recommended step-up dosing and administer concomitant medications before and after Cycle 1 Day 1 and Cycle 1 Day 8 IMDELLTRA infusions as described in Table 3 of the Prescribing Information (PI) to reduce the risk of CRS. Administer IMDELLTRA in an appropriate healthcare facility equipped to monitor and manage CRS. Ensure patients are well hydrated prior to administration of IMDELLTRA.

Closely monitor patients for signs and symptoms of CRS during and after treatment with IMDELLTRA. At the first sign of CRS during treatment, immediately discontinue IMDELLTRA infusion. If CRS occurs during or after treatment, evaluate the patient for hospitalization and manage based on severity. Withhold or permanently discontinue IMDELLTRA based on severity. Counsel patients and caregivers to seek immediate medical attention should signs or symptoms of CRS occur after discharge.

Neurologic Toxicity, Including ICANS: IMDELLTRA can cause life-threatening or fatal neurologic toxicity, including ICANS. In the pooled safety population, neurologic toxicity occurred in 65% of patients who received IMDELLTRA, with Grade 3 or higher events in 7% of patients including fatal events in 0.2%. The most frequent neurologic toxicities were dysgeusia (34%), headache (17%), peripheral neuropathy (9%), dizziness (9%), and insomnia (8%). The incidence of signs and symptoms consistent with ICANS was 10% in IMDELLTRA-treated patients including events with the preferred terms: ICANS (4.7%), muscular weakness (3.2%), cognitive disorder (0.6%), aphasia (0.6%), depressed level of consciousness (0.4%), seizures (0.4%), encephalopathy (0.4%), and leukoencephalopathy (0.2%). There was one fatal reaction of ICANS. Recurrent ICANS occurred in 1.5% of patients. Of the patients who experienced ICANS, most experienced the event following Cycle 1 Day 1 (2.5%) and Cycle 1 Day 8 (3.6%). Following Day 1, Day 8, and Day 15 infusions, 1.3%, 1.3% and 0.4% of patients experienced Grade ≥ 2 ICANS, respectively. ICANS can occur several weeks following administration of IMDELLTRA. The median time to onset of ICANS from the first dose of IMDELLTRA was 16 days (range: 1 to 862 days). The median time to resolution of ICANS was 4 days (range: 1 to 40 days).

The onset of ICANS can be concurrent with CRS, following resolution of CRS, or in the absence of CRS. Clinical signs and symptoms of ICANS may include but are not limited to confusional state, depressed level of consciousness, disorientation, somnolence, lethargy, and bradyphrenia.

Patients receiving IMDELLTRA are at risk of neurologic adverse reactions and ICANS resulting in depressed level of consciousness. Advise patients to refrain from driving and engaging in hazardous occupations or activities, such as operating heavy or potentially dangerous machinery, until neurologic symptoms resolve.

Closely monitor patients for signs and symptoms of neurologic toxicity and ICANS during treatment with IMDELLTRA. At the first sign of ICANS, immediately discontinue the infusion, evaluate the patient and provide supportive therapy based on severity. Withhold IMDELLTRA or permanently discontinue based on severity.

Cytopenias: IMDELLTRA can cause cytopenias including neutropenia, thrombocytopenia, and anemia. In the pooled safety population, based on laboratory data, decreased neutrophils occurred in 16% of patients, including 9% Grade 3 or 4. The median time to onset for Grade 3 or 4 decreased neutrophil count was 41 days (range: 2 to 306 days). Decreased platelets occurred in 30% including 2.2% Grade 3 or 4. The median time to onset for Grade 3 or 4 decreased platelets was 67 days (range: 3 to 420 days). Decreased hemoglobin occurred in 56% of patients, including 4.7% Grade 3 or 4. Febrile neutropenia was reported as an adverse event in 1.5% of patients treated with IMDELLTRA.

Monitor patients for signs and symptoms of cytopenias. Perform complete blood counts prior to treatment with all doses of IMDELLTRA, up through Cycle 5 Day 15 and then prior to administration on Day 1 of each cycle starting with Cycle 6. Based on the severity of cytopenias, temporarily withhold, or permanently discontinue IMDELLTRA.

Infections: IMDELLTRA can cause serious infections, including life-threatening and fatal infections.

In the pooled safety population, infections, including opportunistic infections, occurred in 43% of patients who received IMDELLTRA, including 14% Grade 3 or 4. The most frequent infections were pneumonia (11%), urinary tract infection (9%), COVID-19 (6%), upper respiratory tract infection (4.7%), respiratory tract infection (4%), candida infection (2.1%), oral candidiasis (2.1%), and nasopharyngitis (2.1%).

Monitor patients for signs and symptoms of infection prior to and during treatment with IMDELLTRA and treat as clinically indicated. Withhold or permanently discontinue IMDELLTRA based on severity.

Hepatotoxicity: IMDELLTRA can cause hepatotoxicity. In the pooled safety population, based on laboratory data, elevated ALT occurred in 39% of patients who received IMDELLTRA, including 2.5% with Grade 3 or 4 ALT. Elevated AST occurred in 43% of patients, including 3.2% Grade 3 or 4. Elevated bilirubin also occurred in 16% of patients, including 1.3% Grade 3 or 4. Liver enzyme elevation can occur with or without concurrent CRS.

Monitor liver enzymes and bilirubin prior to treatment with IMDELLTRA, and as clinically indicated. Withhold IMDELLTRA or permanently discontinue based on severity.

Hypersensitivity: IMDELLTRA can cause severe hypersensitivity reactions. Clinical signs and symptoms of hypersensitivity may include, but are not limited to, rash and bronchospasm. Monitor patients for signs and symptoms of hypersensitivity during treatment with IMDELLTRA and manage as clinically indicated. Withhold or consider permanent discontinuation of IMDELLTRA based on severity.

Embryo-Fetal Toxicity: Based on its mechanism of action, IMDELLTRA may cause fetal harm when administered to a pregnant woman. Advise patients of the potential risk to a fetus. Advise females of reproductive potential to use effective contraception during treatment with IMDELLTRA and for 2 months after the last dose.
ADVERSE REACTIONS

The pooled safety population reflects exposure to intravenous IMDELLTRA, as a single agent, at the recommended dosage of IMDELLTRA 1 mg on Cycle 1 Day 1 followed by 10 mg on Days 8 and 15, and then every 2 weeks until disease progression or intolerable toxicity in 473 patients with small cell lung cancer enrolled in three clinical trials: DeLLphi-300, DeLLphi-301 and DeLLphi-304. Among 473 patients who received IMDELLTRA, 40% were exposed for 6 months or longer and 19% were exposed for greater than one year.
The most common (≥ 20%) adverse reactions were CRS (57%), fatigue (48%), decreased appetite (38%), dysgeusia (34%), pyrexia (33%), constipation (31%), musculoskeletal pain (31%), and nausea (25%).
The most common (≥ 5%) Grade 3 or 4 laboratory abnormalities were decreased lymphocytes (43%), decreased sodium (12%), decreased total neutrophils (9%), and increased uric acid (6%).
DOSAGE AND ADMINISTRATION: Important Dosing Information

Administer IMDELLTRA as an intravenous infusion over 1 hour.
Administer IMDELLTRA according to the step-up dose in the IMDELLTRA PI (Table 1) to reduce the incidence and severity of CRS.
Evaluate complete blood count, liver enzymes and bilirubin prior to administration of all doses of IMDELLTRA up through Cycle 5 Day 15 and then prior to administration of IMDELLTRA on Day 1 of each cycle starting with Cycle 6. More frequent evaluation may be necessary if clinically indicated.
For Cycle 1, administer recommended concomitant medications before and after Cycle 1 Day 1 and Cycle 1 Day 8 IMDELLTRA infusions to reduce the risk of CRS reactions as described in the PI (Table 3).
IMDELLTRA should only be administered by a qualified healthcare professional with appropriate medical support to manage severe reactions such as CRS and neurologic toxicity including ICANS.
Due to the risk of CRS and neurologic toxicity, including ICANS, monitor patients from the start of the IMDELLTRA infusion for 6 to 8 hours following Cycle 1 Day 1 and Cycle 1 Day 8 in an appropriate healthcare setting.
Perform patient assessment including vital signs check on Cycle 1 Days 2 and 9.
Recommend that patients remain within 1 hour of an appropriate healthcare setting for a total of 48 hours from the start of the infusion with IMDELLTRA following Cycle 1 Day 1 and Cycle 1 Day 8 doses, accompanied by a caregiver.
Inform both the patient and the caregiver on the signs and symptoms of CRS and ICANS prior to discharge.
Ensure patients are well hydrated prior to administration of IMDELLTRA.

(Press release, Amgen, SEP 14, 2026, View Source [SID1234670839])