On October 14, 2024 Halia Therapeutics, Inc. (Halia), a clinical-stage biopharmaceutical company at the forefront of developing innovative treatments for inflammatory and neurodegenerative diseases, reported that David J. Bearss, Ph.D., President and CEO of Halia Therapeutics, will participate in a panel at the BIO Investor Forum taking place October 15-16, 2024, in San Francisco, CA (Press release, Halia Therapeutics, OCT 14, 2024, View Source [SID1234647180]).
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Dr. Bearss will take part in the panel discussion: ‘Expansion of Neurodegenerative Disease Targets and Pipelines’ which will take place on Tuesday, October 15, 11:00-11:50 am PT at the Venetian Room, Fairmont San Francisco.
This session will feature leading innovators who will discuss the latest advancements in diverse approaches to neurodegenerative diseases. Attendees will learn about the evolving landscape of neurodegenerative disease research and development, the potential of novel targets, and the implications for future treatment and investment opportunities.
A Phase 1 trial evaluating Halia Therapeutics’ compound HT-4253 was recently initiated to assess its safety, tolerability, and pharmacokinetics in healthy adults. HT4253 functions by inhibiting LRRK2, a regulator in neuroinflammation associated with Alzheimer’s and Parkinson’s disease.
Bio Investor Forum showcases drug development programs that are ready for partnering and venture funding, with a focus on treating patients by accelerating the progress of new therapeutic technologies into commercialization. David J. Bearss, Ph.D., President and CEO, will be available for one-on-one investor meetings with registered conference attendees at the forum. Meetings can be scheduled through the partnering system.
To view the full agenda please visit View Source .
About HT-4253
HT-4253 is an orally administered small molecule with excellent brain penetration that targets a mediator of neuroinflammation called LRRK2. Chronic inflammation in the brain is a driver of several neurodegenerative diseases, such as Parkinson’s disease and Alzheimer’s disease. Halia scientists have shown that LRRK2 is an essential regulator of neuroinflammation. LRRK2 inhibition by HT-4253 could represent a new way to treat neurodegenerative diseases with an inflammation component by changing how the disease progresses. HT-4253 is currently being evaluated in a Phase I trial determining its safety and tolerability in healthy volunteer subjects (NCT06537817).